Jonas Olofsson Foto: Psykologiska institutionen/HB

Jonas Olofsson

Docent, universitetslektor

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Arbetar vid Psykologiska institutionen
Telefon 08-16 38 40
Besöksadress Frescati hagväg 9A
Rum 213a
Postadress Psykologiska institutionen 106 91 Stockholm

Om mig

Jag forskar och undervisar om perception, minne och emotion, med särskilt fokus på hur doftsinnet påverkar dessa processer. Jag leder en forskargrupp som undersöker om doftsinnet kan vara en tidig markör för demenssjukdom, varför det är så svårt att namnge dofter, om man kan träna hjärnan med doftbaserade datorspel, och om dofter kan påverka sociala processer. För att besvara dessa frågor mäter vi beteenden och hjärnaktivitet med olika metoder. För en aktuell publikationslista och citeringsindex, se min Google scholar-sida.


Jag disputerade 2008 vid Umeå Universitet och blev docent i psykologi vid Stockholms universitet (SU) 2009. Jag har varit verksam som forskare vid Northwestern University (Chicago, 2009-2011), The Scripps Research Institute (San Diego, 2005-2006) och Karolinska Institutet (Stockholm, 2009). Sedan 2012 är jag vid sidan av min tjänst vid SU även knuten till Swedish Collegium for Advanced Study i Uppsala, och sedan 2016 är jag adjungerad forskare vid New York Universitys medicinska fakultet. Min forskning stöds av Vetenskapsrådet, Riksbankens Jubileumsfond och Marianne och Marcus Wallenbergs fond.


Odor identification in aging and dementia: Influences of cognition and the ApoE gene. Doctoral dissertation from the Department of Psychology, Umeå University. ISBN 978-91-7264-652-0


Antagen 2016 till Wallenberg Academy Fellows, ett karriärprogram för Sveriges mest lovande unga forskare. Programmet omfattar alla ämnesområden.

Invald 2015 i Sveriges Unga Akademi, en organisation för några av landets bästa yngre forskare, som verkar för att förbättra yngre forskares villkor och främja vetenskapens roll i samhället.

Antagen 2012 till Pro Futura Scientia VII, ett nationellt forskningsprogram inrättat för särskilt lovande unga forskare inom humaniora och samhällsvetenskap.

Tilldelades 2010 års pris till unga forskare i psykologi av Nationalkommittén för Psykologi (del av Kungl. Vetenskapsakademien) efter att ha nominerats av Umeå och Stockholms universitet.

Tilldelades 2009 års Fulker award av tidskriften Behavior Genetics för artikeln Odor identification deficit as a predictor of five-year global cognitive change: Interactive effects with age and ApoE-e4, som utvaldes till årets mest förtjänstfulla artikel.


I urval från Stockholms universitets publikationsdatabas
  • 2016. Jonas K. Olofsson (et al.). Neuropsychologia 85, 1-9

    The ɛ4 allele of the Apolipoprotein E gene is a genetic risk factor for late-onset dementia of the Alzheimers' type (DAT), which is characterized by loss of both episodic memoryand olfactory functions. Little is known about the possible role of ɛ4 in the association between ongoing episodic memory decline and olfactory deficits in the general population, but such information is relevant in determining the relevance of olfaction as a marker of DAT risk. The present study was based on a large, population-based sample (n=1087, aged 45–90 years, of which 324 were ɛ4-carriers). Episodic memory change rates were established using data collected every 5 years for a 10–20 year interval leading up to an olfactory assessment using the Scandinavian Odor Identification Test at the last wave of data collection. Participants were classified according to whether or not their episodic memory ability declined more rapidly than the age-typical norm (by >1SD). Our main result is that only in ɛ4-carriers was episodic memory decline associated with odor identification impairment. In individuals without ɛ4, odor identification was unrelated to episodic memory decline status. Follow-up analyses indicated that this moderation by ɛ4 was due to the olfactory nature of the identification test, and that the effect was not caused by 63 individuals with dementia. Our results suggest that the ɛ4 determines the functional association between ongoing episodic memory decline and olfaction. These findings are consistent with the notion that ɛ4-carriers with DAT, compared to non-carriers, display a cortical atrophy pattern that is more focused on mediotemporal lobe regions supporting olfactory and episodic memory functions. Olfactory and memory assessments might provide complementary information on mediotemporal atrophy prior to clinical dementia onset, but the ɛ4 should be considered when using olfactory assessment as an early-stage indicator.

  • 2016. Maria Larsson (et al.). 17th International Symposium on Olfaction and Taste (ISOT2016), Yokohama, Japan, June 5-9, 2016. Chemical Senses, 41(9), E216

    The objective of this study was to examine the association between performance in odor identification and future mortality in a community cohort of adults aged between 40 and 90 years. We assessed olfactory performance with a 13-item-version of the Scandinavian Odor Identification Test (SOIT). The results showed that during follow-up (mean=9.4 years, standard deviation=2.23), 411 of 1774 (23.2%) participants died. In a Cox model, the association between higher SOIT score and mortality was highly significant (hazard ratio [HR]=0.74, per point interval, 95% confidence interval [CI]=0.71–0.77, p<0.001). The effect was attenuated, but remained significant after controlling for age, sex, education, and health and cognitive variables that were also associated with an increased risk of mortality (HR=0.92, 95% CI=0.87–0.97, p=0.001). Controlling for dementia conversion prior to death did not attenuate the association between SOIT score and mortality (HR=0.92, 95% CI=0.87–0.97, p=0.001). Similar results were obtained for olfactory sensitivity as assessed by self-report. Overall, the present findings show that poor odor identification performance is associated with an increased likelihood of future mortality in middle-aged and older adults, after controlling for social, cognitive, and medical risk factors. Most importantly, controlling for the development of dementia did not attenuate the association between odor identification and mortality, suggesting that olfactory decline might mark deteriorating health also irrespective of dementia.

  • 2016. Veit Kubik (et al.). Journal of Cognitive Psychology 28 (2), 209-219

    Testing memory typically enhances subsequent re-encoding of information (“indirect” testing effect) and, as compared to restudy, it also benefits later long-term retention (“direct” testing effect). We investigated the effect of testing on subsequent restudy and 1-week retention of action events (e.g. “water the plant”). In addition, we investigated if the type of recall practice (noun-cued vs. verb-cued) moderates these testing benefits. The results showed an indirect testing effect that increased following noun-cued recall of verbs as compared to verb-cued recall of nouns. In contrast, a direct testing effect on the forgetting rate of performed actions was not reliably observed, neither for noun- nor verb-cued recall. Thus, to the extent that this study successfully dissociated direct and indirect testing-based enhancements, they seem to be differentially effective for performed actions, and may rely on partially different mechanisms.

  • 2016. Petter Kallioinen (et al.). Frontiers in Psychology 7

    Difficulties in auditory and phonological processing affect semantic processing in speech comprehension for deaf and hard-of-hearing (DHH) children. However, little is known about brain responses related to semantic processing in this group. We investigated event-related potentials (ERPs) in DHH children with cochlear implants (CIs) and/or hearing aids (HAs), and in normally hearing controls (NH). We used a semantic priming task with spoken word primes followed by picture targets. In both DHH children and controls, cortical response differences between matching and mismatching targets revealed a typical N400 effect associated with semantic processing. Children with CI had the largest mismatch response despite poor semantic abilities overall; Children with CI also had the largest ERP differentiation between mismatch types, with small effects in within-category mismatch trials (target from same category as prime) and large effects in between-category mismatch trials (where target is from a different category than prime), compared to matching trials. Children with NH and HA had similar responses to both mismatch types. While the large and differentiated ERP responses in the CI group were unexpected and should be interpreted with caution, the results could reflect less precision in semantic processing among children with CI, or a stronger reliance on predictive processing.

  • 2015. Jonas Olofsson, Johan Örestig.
Visa alla publikationer av Jonas Olofsson vid Stockholms universitet

Senast uppdaterad: 21 november 2018

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